Golf Course Management

JUL 2013

Golf Course Management magazine is dedicated to advancing the golf course superintendent profession and helping GCSAA members achieve career success.

Issue link: http://gcmdigital.gcsaa.org/i/139656

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research Effect of preemergence herbicides on bermudagrass recovery from SDS Be careful what you spray: Some pre-emergence herbicides may limit turf recovery and further weaken the turfgrass system. Spring dead spot (SDS) is a soil-borne disease caused by three closely related ectotrophic, rootinfecting pathogens: Ophiosphaerella narmari, O. korrae and O. herpotricha (1). Spring dead spot symptoms may occur on intensely managed home lawns, athletic felds, golf courses and sod farms located in the transition zone where winter temperatures are cold enough to induce bermudagrass dormancy (16,20). Disease symptoms are most noticeable when bermudagrass breaks winter dormancy in the spring, but injury may persist into late summer L.L. Beck, Ph.D. T. Cooper A.J. Hephner C.M. Straw G.M. Henry, Ph.D. 80 GCM July 2013 (7). Symptoms appear as well-defned, bleached, circular patches that range in size from a few inches to a yard in diameter (1,7). Turfgrass plants within the disease patches eventually collapse to the ground, leaving behind sunken necrotic areas (21). The germination of weeds in the center of disease patches may cause a decline in turfgrass aesthetic quality and reduce the playability of turf. In severe cases, bermudagrass may not fully recover from spring dead spot symptoms before the onset of winter, leaving the turf susceptible to further damage from freezing temperatures (14). Exposure to dinitroaniline herbicides may cause abnormal swelling ("clubbing") of bermudagrass roots. Photo by Jim Brosnan

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